By Kenneth Blum
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Extra resources for Alcohol and Opiates. Neurochemical and Behavioral Mechanisms
Acetaldehyde, an inhibitor of the enzymatic oxidation of 5-hydroxyindoleacetaldehyde. Biochem. Pharm. 25:535-538 (1969). , Ungar, F. and Alivisatos, S. G. : Aldehyde derivatives of indoleamines and the enhancement of their binding onto brain macromolecules by pentobarbital and acetaldehyde. Nature New Biology 238(82): 126-128 (1972). Korsten, M. , Lieber, C. : High blood acetaldehyde levels after ethanol administra tion difference between alcoholic and non-alcoholic sub jects. New Eng. J. Med.
The contradictions within these studies are probably due to the nonequivalence of the symp toms examined by the various authors (21,23). Thus, while the studies of Goldstein (21) and Blum (22) indicated that catecholaminergic neurons may play a determinant role in producing convulsions due to handling (11) in mice undergoing withdrawal, the studies of Collier (23) indicate a primary role for serotonergic systems in determining withdrawal symptomatology. However, Collier (23) monitored head twitch ing as a primary measure of withdrawal.
Biochem. Pharmacol. 15:711-721 (1966). Videla, L. : Factors that modify the metabolism of ethanol in rat liver and adaptive changes produced by its chronic administration. Biochem. J. 110:275-281 (1970). Pohorecky, L. : Effects of ethanol on central and brain and heart. Acta. 34. 6 A:97- 109 (1968). Aghajanian, G. , Graham, A. W. and Sheard, M. : Serotonin-containing neurons in brain: Depression of firing by MAO inhibitors. Science 169:1100-1102 (1970). Hunt, W. A. : Alterations in the turnover of brain norepinephrine and dopamine in alcohol-dependent rats.