Ciba Foundation Symposium 206 - The Rising Trends in Asthma

Bronchial asthma is a transforming into illness in the course of the constructed international. This quantity provides a severe assessment of all of the attainable components for this emerging development and comprises study that has no longer but been released within the clinical literature. Discusses the elemental biology of bronchial asthma and addresses genetic impacts. Surveys the epidemiological facts for the global tendencies in morbidity and mortality.

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One possible explanation for the temporal differences in these T cell responses, as speculated earlier (Holt et a1 1995), may be the presence or absence of relevant antigen in the fetal environment. Thus, the fetal circulation normally contains IgG subclass antibodies against all the major environmental allergens, and also against tetanus. In the case of dietary and inhalant allergens, exposure of the mother during pregnancy is unavoidable, leading to the possibility of transplacental leakage of allergen fragments with the potential to generate antigen-IgG complexes (nature's archetypal immunogen) in the fetus -this would occur only rarely in the case of tetanus, as vaccination is not normally carried out during pregnancy.

In: Global Initiative for Asthma: global strategy for asthma management and prevention (National Institutes of Health, National A DISEASE OF INFLAMMATION AND REPAIR 27 Heart, Lung and Blood Institute and World Health Organisation Workshop Report). NIH publication no. 53659 Okayama Y, Petit-Frtre C, Kassel 0 et a1 1995 Expression of messenger RNA for IL-4 and IL-5 in human lung and skin mast cells in response to FCE receptor cross-linkage and the presence of stem cell factor. J Immunol 155:17961808 Okayama Y, Lau LC-K, Church MK 1997 TNFa production by human lung mast cells in response to stimulation by stem cell factor and Fc,Rl cross-linkage.

IFNg, y-interferon; IL-4, interleukin 4. tetanus toxoid which did not elicit responses in either cord blood or 10-week-oldinfant samples but (as predicted) did trigger lymphoproliferation in a high proportion of vaccinated infants up to six months old (Holt et al 1995). One possible explanation for the temporal differences in these T cell responses, as speculated earlier (Holt et a1 1995), may be the presence or absence of relevant antigen in the fetal environment. Thus, the fetal circulation normally contains IgG subclass antibodies against all the major environmental allergens, and also against tetanus.

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