Ciba Foundation Symposium - Aetiology of Diabetes Mellitus

Content:
Chapter 1 Chairman's commencing feedback (pages 1–4): Professor R. E. Tunbridge
Chapter 2 Diabetes in a Village neighborhood (pages 5–24): Joan B. Walker
Chapter three Spontaneous Diabetes Mellitus within the chinese language Hamster, Cricetulus griseus. IV. Genetic features (pages 25–48): George Yergaman
Chapter four The Endocrine Pancreas in Human and Experimental Diabetes (pages 49–74): Robertson F. Ogilvie
Chapter five Pancreatic Beta cellphone (pages 75–88): Paul E. Lacy
Chapter 6 Panel dialogue on Biosynthesis and Secretion of Insulin: Biosynthesis of Insulin (pages 89–94): ok. W. Taylor
Chapter 7 Insulin Biosynthesis and Glucose Metabolism in Islet arrangements (pages 95–98): Albert E. Renold
Chapter eight Secretion of Insulin brought about via Leucine in fit matters (pages 99–106): Stefan S. Fajans, John C. Floyd, Jr. Ralph F. Knopf and Jerome W. Conn
Chapter nine fee of free up of Insulin in vitro (pages 107–121): P. J. Randle
Chapter 10 Immunological experiences with Homologous and Heterologous Pancreatic Insulin within the Cow (pages 122–139): Albert E. Renold, Stuart Soeldner and Jurgen Steinke
Chapter eleven Plasma Insulin in Diabetes (pages 140–155): C. N. Hales
Chapter 12 Insulin Antagonism: basic concerns (pages 156–191): David M. Kipnis and Martin F. Stein
Chapter thirteen The Glucose Fatty Acid Cycle and Diabetes Mellitus (pages 192–216): P. J. Randle, P. B. Garland, C. N. Hales and E. A. Newsholme
Chapter 14 Synalbumin Insulin Antagonism and Diabetes (pages 217–239): J. Vallance?Owen
Chapter 15 Pituitary Polypeptide Antagonists of Insulin (pages 240–249): J. Bornstein, Deirdre Hyde and ok. J. Catt
Chapter sixteen Peripheral motion of Insulin Diabetes Mellitus and its Relation to the Aetiology of Diabetes Mellitus (pages 250–281): W. J. H. Butterfield
Chapter 17 The Biochemistry of Blood Vessels (pages 282–300): Helen Muir
Chapter 18 The Glomerular Lesion in Idiopathic and Secondary Diabetes (pages 301–314): M. okay. Macdonald and J. T. Ireland
Chapter 19 Pathophysiology of Microangiopathy in Diabetes Mellitus (pages 315–344): Searle B. Rees, Rafael A. Camerini?Davalos, James B. Caulfield, Oscar Lozano?Castaneda, Alfredo Cervantes?Amezcus, Jan Taton, Daniel Pometta, Jorge P. Krauthammer and Alexander Marble
Chapter 20 Panel dialogue at the function of the Pituitary in Human Diabetes and its problems within the Aetiology of Diabetes Mellitus: The Anterior Pituitary (pages 345–347): J. Bornsteiy
Chapter 21 The Roe of the Pituitary in Human Diabetes (pages 348–353): R. Luft
Chapter 22 The impression of Yttrium?90 Pituitary Implantation on Diabetic Retinopathy (pages 354–390): G. F. Joplin
Chapter 23 ultimate feedback (pages 391–392):

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Additional info for Ciba Foundation Symposium - Aetiology of Diabetes Mellitus and its Complications (Colloquia on Endocrinology), Volume 15

Example text

If an 34 G. YERGANIAN incomplete dominant iiiutation was involved, a comparison of type (a) with type (b) and (c) matings would, in turn, provide some indication of a differential in the expression of homozygotes versus heterozygotes. A total of 390 F, were weaned from approximately 75 type (b) and (c) matings. Contrary to expectancies based upon a recessive gene hypothesis, only three animals developed diabetes ; the remainder were normal in every respect. Diabetes flourished during the course of inbreeding parental lines.

Last summer (1962) the air-conditioning equipment failed to operate properly. Consequently, the temperature and relative humidity persisted for months above 75’F and 75 per cent respectively. The surprising result was that normal animals failed to breed readily, whereas the diabetics responded most favourably. This “negative” experience has helped us to consider the fLiture need for small animal rooms in which the physical environmental factors may be governed to provide optimum controls for the breeding of diabetic animals.

Let me give an example. Ten apparently healthy children of a known diabetic father were tested. /Ioo ml. /Ioo ml. /Ioo ml. at two hours during the glucose tolerance test. /Ioo ml. /Ioo ml. These last three individuals were aglycosuric during the glucose tolerance test. This brings out a very important point, namely that glycosuria as a screening procedure misses many latent, asymptomatic diabetics. In this family there are all stages ofrecognizable diabetes in individuals aged from I 1-30. Thus the disease may have a very slow evolution, but it is not always that slow.

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